Jesse E. Taber, M.D.

Jesse E. Taber, M.D.

Jesse E. Taber, M.D.

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Personal Bio

Treatment Philosophy

Decision making about treatments and testing always requires I have in depth understanding of my patients’ concerns and goals, and that they understand mine. Rapid transmission of information is also critical, which means that at the end of each office visit my patients and their doctors receive a copy of my report and that test results are given as quickly as possible.

Personal Interests

I enjoy spending time with family, playing jazz guitar and piano, karate, cooking, and reading.

Conditions & Procedures


Abnormal Magnetic Resonance Imaging (MRI), Anosmia , Back Pain, Benign Paroxysmal Positional Vertigo (BPPV), Cerebral Palsy (CP) ( ages 18+ years), Cervicogenic Headaches, Chiari Malformation, Cluster Headache, Complicated Migraines, Degenerative Spine Disease, Dizziness, Dysarthria, Dysphagia, Epilepsy/Seizure Disorder, Facet Pain, Facial Pain, Fibromyalgia, Gait Abnormailty, Gait Issues, General Neurology, General Weakness, Head Pain, Headache, Hypoglossal Nerve Disorder, Low Back Pain (LBP), Migraine Headaches, Neck Pain, Occupational Injury, Ocular Migraines, Pain in Limbs, Parasthesia, paresthesias, Spastic Paraparesis, Spasticity, Spinal Stenosis, Syncope, Trigeminal Neuralgia, Twitch, Vertigo, Visual Auras

General Information




NorthShore Medical Group


Neurology, Epilepsy

Academic Rank

Clinical Assistant Professor



Board Certified

Clinical Neurophysiology, Electrodiagnostic Medicine, Neurology

Clinical Service

Education, Training & Fellowships

Medical School

University of Illinois-Chicago, 1985


Weiss Memorial Hospital, 1986


University of Illinois at Chicago, 1989


University of Illinois at Chicago, 1990



NorthShore Medical Group

1000 Central St.
Suite 880
Evanston, IL 60201
847.570.2570 847.926.5353 fax Get Directions This location is wheelchair accessible.

NorthShore Medical Group

9650 Gross Point Rd.
Suite 3900
Skokie, IL 60076
847.570.2570 847.926.5353 fax Get Directions This location is wheelchair accessible.



  • Computerised evaluation of cognitive and motor function.

    Medical & biological engineering & computing 2000 Jan

    Authors: Kisacanin B, Agarwal GC, Taber J, Hier D
    In this paper, we present a clinical study of computerised tracking in the evaluation of cognitive and motor function. We investigate its use in the assessment of effectiveness of antiepileptic drugs (AEDs) as well as in the process of following the progress of Alzheimer's disease (AD). To simplify the experiments, we introduce real-time adaptation of the target speed. In the study with epileptic patients, three result groups are compared: blood levels of AEDs, scores on standard neuropsychological tests, and scores on computerised tracking and reaction time tests. It is found that the computerised tests are repeatable, reliable and sensitive and may therefore be useful in the evaluation of epilepsy treatment. For example, while the blood levels associated with AEDs lie in the therapeutic range, variations in the optimal speed (OS) between 0.9 and 1.1 (expressed in relative units) are recorded. To significantly simplify the protocol for AD patients while preserving its main features, we introduce signal-processing techniques into the data analysis. Local signal property characteristics for AD are found which indicate that the preview tracking of an AD patient is similar to the non-preview tracking of a healthy control. This result is expected since the working memory, which is involved in movement planning, is impaired in AD. In non-preview tracking, healthy control subjects are mostly in tracking mode 1 and have a mean mode duration of 600 ms. In preview tracking, AD patients are mostly in mode 2 with a mean mode duration of 600 ms.
    PMID: 10829393 [PubMed - as supplied by publisher]
  • TRI-PLEDs: a case report.

    Clinical EEG (electroencephalography) 1998 Apr

    Authors: Hughes JR, Taber J, Uppal H
    This case report shows an example of TRI-PLEDs, periodic discharges occurring independently on 3 different areas. The phenomenon of the PLED is briefly discussed.
    PMID: 9571299 [PubMed - as supplied by publisher]
  • Cranial computed tomographic observations in multi-infarct dementia. A controlled study.

    Stroke; a journal of cerebral circulation 1992 Jun

    Authors: Gorelick PB, Chatterjee A, Patel D, Flowerdew G, Dollear W, Taber J, Harris Y
    We compared cranial computed tomography findings among 58 multi-infarct dementia index cases and 74 multi-infarct control subjects without cognitive impairment to identify potential determinants of multi-infarct dementia.
    The cranial computed tomography records of acute ischemic stroke patients with a history of multiple cerebral infarcts were compared to determine the number, location, and size of cerebral infarcts; the pattern of infarction; brain volume loss; and the degree of white matter lucency, sulcal enlargement, and ventricular enlargement. Multi-infarct patients were divided into two groups: 1) index cases were defined as those with multi-infarct dementia as defined by the Diagnostic and Statistical Manual of Mental Disorders, edition 3 (DSM-III) criteria; and 2) control subjects were defined as those multi-infarct patients without dementia or multi-infarct dementia according to DSM-III criteria.
    Overall, multi-infarct index cases had more cerebral infarcts, more cortical and subcortical left hemisphere infarcts, higher mean ventricular volume to brain volume ratio, more extensive enlargement of the body of the lateral ventricles and cortical sulci, and a higher prevalence of white matter lucencies. Among multi-infarct cases and control subjects the most frequent site of infarction was the subcortical region, and the most frequent pattern of infarction was lacunar. Stepwise logistic regression analysis examined cranial computed tomography as well as other factors and showed that level of education, stroke severity, left cortical infarction, and diffuse enlargement of the left lateral ventricle were the best overall predictors of multi-infarct dementia.
    Level of education, stroke severity, and left hemisphere infarction may be predictors of multi-infarct dementia.
    PMID: 1595096 [PubMed - as supplied by publisher]
  • The effect of spikes and spike-free epochs on topographic brain maps.

    Clinical EEG (electroencephalography) 1991 Jul

    Authors: Hughes JR, Taber JE, Fino JJ
    Maps of foci, when no discharges nor obvious slow activity were included in the map, usually (84%) showed changes within the focus itself and also in adjacent areas (58%) and often (47%) in the mirror region. An increase in activity was more often seen in the more active foci, especially in the delta and beta 2 ranges, and decrease in activity was usually seen with inactive foci. When discharges were included in the maps, an increasing number was associated with an increase at the main focus in all ranges, especially in delta and beta 1, but much less often in beta 2. The amount of electrical charge required for a change in the map with spikes was approximately 40 nanocoulombs (nC) for the first and second changes, increasing to around 50 nC with adjacent or separate foci. "Artificial" spikes produce more delta activity than other frequency ranges and the FFT of various pulses show maximal amplitudes at the slowest frequencies.
    PMID: 1879054 [PubMed - as supplied by publisher]
  • Clinical and angiographic comparison of asymptomatic occlusive cerebrovascular disease.

    Neurology 1988 Jun

    Authors: Gorelick PB, Caplan LR, Langenberg P, Hier DB, Pessin M, Patel D, Taber J
    We compared clinical and arteriographic features in 106 patients with symptomatic unilateral carotid territory occlusive disease to determine the frequency and distribution of occlusive arterial lesions in asymptomatic vessels. Among black patients who were predominantly from Chicago, young, and female, there were fewer transient ischemic attacks and myocardial infarcts, less claudication, and more asymptomatic lesions of the supraclinoid internal carotid artery, anterior cerebral artery stem, and the middle cerebral artery stem. Among white patients predominantly from New England, elderly, and male, there was more frequent and severe occlusive asymptomatic disease at extracranial carotid and vertebral artery sites. Knowledge of the distribution of asymptomatic lesions will help guide evaluation and treatment strategies for patients with occlusive cerebrovascular disease.
    PMID: 3368065 [PubMed - as supplied by publisher]