Hamad I. Farhat, M.D.

Hamad I. Farhat, M.D.

Hamad I. Farhat, M.D.

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Conditions & Procedures

Conditions

Acoustic Neuroma, Aneurysm, Arterio-Venous Malformation (AVM), astrocytoma, Atypical Facial Pain, Back Pain, Brain Bleed, Brain Tumor, Cerebral Ischemia, Cerebral Vasculitis, Cerebrospinal Fluid (CSF) Leak, Cerebrovascular Accident (CVA), Cervical/Lumbar Disc Disease, Chiari Malformation, Chordoma, Cranial Base Tumors, Craniopharyngioma, Degenerative Spine Disease, Ependynoma, Glioblastoma, Glioma, Hemifacial Spasm, Hydrocephalus, Idiopathic Intracranial Hypertension, Meningioma, Neoplasm, Normal Pressure Hydrocephalus, Pineal Cyst, Pituitary Adenoma (Micro or Macro- Adenoma), Pituitary Tumor, Pseudo Tumor Cerebri, Radiculopathy, Rathke's Cleft Cyst, Spinal Cord Tumors, Spinal Fractures, Spinal Stenosis, Subarachnoid Hemorrhage (SAH), Subdural Hematoma (SDH), Synovial Cysts, Syringomyelia, Trigeminal Neuralgia, Vestibular Schwanoma

Procedures

Cerebral Embolization, Cervical and Lumbar Surgery, Epilepsy Surgery, Stereotactic Neurosurgery and Radiosurgery (SRS), Stereotactic Radiosurgery of Spine, Vascular Surgery

General Information

Gender

Male

Affiliation

NorthShore Medical Group

Academic Rank

Clinical Assistant Professor

Languages

Arabic, English, French, Spanish

Clinical Service

Education, Training & Fellowships

Medical School

Chicago Medical School/Finch University of Health, 2003

Internship

Jackson Memorial Hospital/Univ of Miami School of Medicine, 2004

Residency

Jackson Memorial Hospital/Univ of Miami School of Medicine, 2010

Fellowship

Jackson Memorial Hospital/Univ of Miami School of Medicine, 2008

Locations

A

NorthShore Medical Group

1000 Central St.
Suite 880
Evanston, IL 60201
847.570.1440 847.570.1442 fax This location is wheelchair accessible.

Insurance

Every effort has been made to ensure the accuracy of the information in this directory. However, some changes may occur between updates. Please check with your provider to ensure that he or she participates in your health plan.

Aetna HMO/PPO/POS
BCBS HMOI
BCBS PPO *except Blue Choice IL
Beechstreet PPO
CCN PPO
CIGNA Choice Fund
CIGNA Choice Fund PPO
CIGNA EPO
CIGNA Network
CIGNA Network Open Access
CIGNA POS
CIGNA POS Open Access
CIGNA PPO
CIGNA:Open Access Plus
Community Care Partners
First Health PPO
Galaxy PPO
Great West POS
Great West PPO
Healthcares Finest Network PPO
Humana Choice Care PPO
Humana IPA--HMO
Humana POS
Humana PPO
Land of Lincoln
Medicare
Multiplan Admar PPO
Multiplan Formost PPO
Multiplan Health Network PPO
Multiplan Wellmark PPO
PHCS PPO
Preferred Plan PPO
Railroad Medicare - Cook County
Railroad Medicare - Lake County
UHC *except Core & Navigate
Unicare PPO

Publications

  • The Early Versus Late MR Debate.

    World neurosurgery 2014 Aug 25

    Authors: Shakur SF,
    Abstract
    Glioblastoma (GBM) rarely presents as an infratentorial tumor in adults. The authors present a case of concomitant supratentorial and infratentorial GBM in an adult. A 72-year-old man presented with headache, nausea, vomiting, and lightheadedness. Initial MR images revealed enhancing masses in the right cerebellum and right posterior periventricular region. The patient underwent a suboccipital craniotomy and resection of the cerebellar lesion. Final histopathology was consistent with glioblastoma. The patient went on to receive standard radiation treatment for GBM with concurrent and adjuvant temozolomide. However, the patient experienced clinical deterioration within a few days after starting radiotherapy. He and his family decided to forego treatment and pursue palliative care. The patient expired three months after the initial diagnosis. Autopsy findings supported the diagnosis of GBM with leptomeningeal gliomatosis and involvement of the cerebrum, cerebellum, and spinal cord. The authors review the literature and propose that the pathogenesis of multiple and multicentric GBM may involve neural stem cells within the subventricular zone or could result from tumor dissemination along established CNS routes, such as white matter tracts and CSF pathways.
    PMID: 25169747 [PubMed - as supplied by publisher]
  • Charcot-Marie-Tooth and trigeminal neuralgia.

    Clinical neurology and neurosurgery 2013 Oct

    Authors: Wong RH,
    Abstract
    Asymptomatic solitary meningiomas are typically managed with clinical and radiographic follow-up. Multiple meningiomas represents a clinical entity distinct from solitary meningiomas and can be sporadic, radiation-induced, associated with neurofibromatosis, or exhibit other familial inheritance. The growth rate for multiple meningiomas is not known and therefore management of these complicated patients can be difficult.
    A retrospective chart review was performed on 12 patients with a total of 55 meningiomas. Patients with neurofibromatosis were not included. Serial enhanced magnetic resonance imaging was used to determine tumor growth rates. Treatment history was also reviewed and included for analysis.
    Analysis of all 55 tumors demonstrated an average rate of growth of 0.46 cm(3)/year (range: -0.57-2.94 cm(3)/year). In the 23 tumors that received no treatment, the average rate of growth was 0.34 cm(3)/year (range: -0.03-1.8 cm(3)/year). Ten of the 23 tumors that received no treatment had no history of cranial irradiation. This group demonstrated a growth rate of 0.44 cm(3)/year (range: -0.01-1.8 cm(3)/year). Linear regression analysis did not yield any significant relationship between tumor burden and rates of growth.
    Tumor growth rates in patients with multiple meningiomas did not appear to be higher than reported rates for incidentally found solitary meningiomas. As such, asymptomatic multiple meningioma patients should be managed with clinical and radiographic follow-up.
    PMID: 23911003 [PubMed - as supplied by publisher]
  • IN VITRO QUANTIFICATION OF THE SIZE DISTRIBUTION OF INTRASACCULAR VOIDS LEFT AFTER ENDOVASCULAR COILING OF CEREBRAL ANEURYSMS.

    Cardiovascular engineering and technology 2013 Mar 1

    Authors: Sadasivan C,
    Abstract
    Endovascular coiling of cerebral aneurysms remains limited by coil compaction and associated recanalization. Recent coil designs which effect higher packing densities may be far from optimal because hemodynamic forces causing compaction are not well understood since detailed data regarding the location and distribution of coil masses are unavailable. We present an in vitro methodology to characterize coil masses deployed within aneurysms by quantifying intra-aneurysmal void spaces.
    Eight identical aneurysms were packed with coils by both balloon- and stent-assist techniques. The samples were embedded, sequentially sectioned and imaged. Empty spaces between the coils were numerically filled with circles (2D) in the planar images and with spheres (3D) in the three-dimensional composite images. The 2D and 3D void size histograms were analyzed for local variations and by fitting theoretical probability distribution functions.
    Balloon-assist packing densities (31±2%) were lower (p=0.04) than the stent-assist group (40±7%). The maximum and average 2D and 3D void sizes were higher (p=0.03 to 0.05) in the balloon-assist group as compared to the stent-assist group. None of the void size histograms were normally distributed; theoretical probability distribution fits suggest that the histograms are most probably exponentially distributed with decay constants of 6-10 mm. Significant (p<=0.001 to p=0.03) spatial trends were noted with the void sizes but correlation coefficients were generally low (absolute r<=0.35).
    The methodology we present can provide valuable input data for numerical calculations of hemodynamic forces impinging on intra-aneurysmal coil masses and be used to compare and optimize coil configurations as well as coiling techniques.
    PMID: 23687520 [PubMed - as supplied by publisher]
  • Transplanum approach to suprasellar lesions.

    World neurosurgery

    Authors:
    Abstract
    PMID: 23500340 [PubMed - as supplied by publisher]
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