Mark K. Bowen, M.D.

Mark K. Bowen, M.D.

Mark K. Bowen, M.D.

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Personal Bio

Treatment Philosophy

My goal is to provide the highest level, state-of-the-art care of knee and shoulder injuries and conditions in a timely, caring and compassionate manner.

Personal Interests

I enjoy time with my family as well as cycling, travel and photography.

Conditions & Procedures

Conditions

Knee Injury, Shoulder Injury

Procedures

ACL Reconstruction, Arthroscopy

General Information

Gender

Male

Affiliation

NorthShore Medical Group

Expertise

ACL Tears, Rotator Cuff Tears, Meniscus Tears

Languages

English

Board Certified

Orthopaedic Surgery

Clinical Service

Sports Medicine

Education, Training & Fellowships

Medical School

Cornell University Medical College, 1985

Internship

New York Hospital/Weill Cornell Univ Schl of Medicine, 1986

Residency

Hospital for Special Surgery, 1990

Fellowship

Hospital for Special Surgery, 1991

Locations

A

NOI NorthShore Orthopedics Chicago

680 N Lake Shore Dr
Ste 924
Chicago, IL 60611
847.866.7846 866.954.5787 fax This location is wheelchair accessible.
B

NorthShore Medical Group

9650 Gross Point Rd.
Suite 2900
Skokie, IL 60076
847.866.7846 866.954.5787 fax This location is wheelchair accessible.
C

NOI NorthShore Orthopedics Glenview

2501 Compass Rd
Ste 125
Glenview, IL 60026
847.866.7846 866.954.5787 fax This location is wheelchair accessible.

Insurance

Every effort has been made to ensure the accuracy of the information in this directory. However, some changes may occur between updates. Please check with your provider to ensure that he or she participates in your health plan.

Aetna HMO/PPO/POS
BCBS HMOI
BCBS PPO *except Blue Choice IL
Beechstreet PPO
CCN PPO
CIGNA Choice Fund
CIGNA Choice Fund PPO
CIGNA EPO
CIGNA Network
CIGNA Network Open Access
CIGNA POS
CIGNA POS Open Access
CIGNA PPO
CIGNA:Open Access Plus
First Health PPO
Galaxy PPO
Great West POS
Great West PPO
Healthcares Finest Network PPO
Humana Choice Care PPO
Humana IPA--HMO
Humana POS
Humana PPO
Land of Lincoln
Medicare
Multiplan Admar PPO
Multiplan Formost PPO
Multiplan Health Network PPO
Multiplan Wellmark PPO
NorthShore Employee Network I (EPO Option)
NorthShore Employee Network II (EPO Plus & CDHP)
PHCS PPO
Preferred Plan PPO
Railroad Medicare - Cook County
Railroad Medicare - Lake County
UHC *except Core & Navigate
Unicare PPO

Publications

  • Acromioclavicular joint injuries in the national football league: epidemiology and management.

    The American journal of sports medicine 2013 Dec

    Authors: Lynch TS,
    Abstract
    Previous studies investigating acromioclavicular (AC) joint injuries in professional American football players have only been reported on quarterbacks during the 1980s and 1990s. These injuries have not been evaluated across all position players in the National Football League (NFL).
    The purpose of this study was 4-fold: (1) to determine the incidence of AC joint injuries among all NFL position players; (2) to investigate whether player position, competition setting, type of play, and playing surface put an athlete at an increased risk for this type of injury; (3) to determine the incidence of operative and nonoperative management of these injuries; and (4) to compare the time missed for injuries treated nonoperatively to the time missed for injuries requiring surgical intervention.
    Descriptive epidemiological study.
    All documented injuries of the AC joint were retrospectively analyzed using the NFL Injury Surveillance System (NFLISS) over a 12-season period from 2000 through 2011. The data were analyzed by the anatomic location, player position, field conditions, type of play, requirement of surgical management, days missed per injury, and injury incidence.
    Over 12 NFL seasons, there were a total of 2486 shoulder injuries, with 727 (29.2%) of these injuries involving the AC joint. The overall rate of AC joint injuries in these athletes was 26.1 injuries per 10,000 athlete exposures, with the majority of these injuries occurring during game activity on natural grass surfaces (incidence density ratio, 0.79) and most often during passing plays. These injuries occurred most frequently in defensive backs, wide receivers, and special teams players; however, the incidence of these injuries was greatest in quarterbacks (20.9 injuries per 100 players), followed by special teams players (20.7/100) and wide receivers (16.5/100). Overall, these athletes lost a mean of 9.8 days per injury, with quarterbacks losing the most time to injury (mean, 17.3 days). The majority of these injuries were low-grade AC joint sprains that were treated with nonoperative measures; only 13 (1.7%) required surgical management. Players who underwent surgical management lost a mean of 56.2 days.
    Shoulder injuries, particularly those of the AC joint, occur frequently in the NFL. These injuries can result in time lost but rarely require operative management. Quarterbacks had the highest incidence of injury; however, this incidence is lower than in previous investigations that evaluated these injuries during the 1980s and 1990s.
    PMID: 24057030 [PubMed - as supplied by publisher]
  • Arthroscopic treatment of acromioclavicular joint injuries and results.

    Clinics in sports medicine 2003 Apr

    Authors: Nuber GW,
    Abstract
    Injuries and conditions that affect the AC joint are common. Low-grade separations, degenerative conditions, and osteolysis of the distal clavicle are frequently dealt with by the treating physician. Proper assessment requires a thorough history, examination, and radiologic work-up. An injection of bupivicaine into the AC joint can be a very useful test to evaluate the source of pain about the symptomatic shoulder. Most conditions affecting the AC joint can be treated conservatively, but patients who do not respond to these treatments or athletes who do not wish to modify their activities may require resection of the distal clavicle and the AC joint. Operative intervention can be performed as an open procedure with good results. Recent advances in operative arthroscopic procedures allow us to replicate and exceed the results of the open resection. Arthroscopic resection can be undertaken via a direct approach that does not violate the subacromial space or via an indirect or bursal approach. The indirect approach allows you to assess both the subacromial space and the AC joint because impingement pathology and subacromial compromise are frequently associated with AC change. The advantage of an arthroscopic resection is its ability to be performed as an outpatient procedure with less compromise of musculotendinous structures, shorter rehabilitation, and quicker return to activity. The amount of bone resection necessary is less than with the open procedure because of the ability to preserve the stabilizing properties of the superior AC ligaments. Resection of 4 mm to 8 mm of bone is all that may be required to give uniformly good results. Arthroscopic resection of the distal clavicle is technically demanding and requires skill and familiarity with other arthroscopic shoulder procedures. Complications related to this procedure are relatively infrequent and include infection, residual pain, lack of adequate bone resection, and instability, particularly in patients with previous grade 1 and 2 separations. Less commonly noted is the symptomatic development of heterotopic bone. To the accomplished arthroscopic shoulder surgeon, arthroscopic resection of the symptomatic AC joint gives excellent clinical results that allow a compromised athlete a relatively quick return to desired sport activities.
    PMID: 12825532 [PubMed - as supplied by publisher]
  • Multiaxis muscle strength in ACL deficient and reconstructed knees: compensatory mechanism.

    Medicine and science in sports and exercise 2002 Jan

    Authors: Zhang LQ,
    Abstract
    It is unclear how muscle strength in tibial rotation and knee abduction change following anterior cruciate ligament (ACL) injury and reconstruction. Such strength changes are likely, considering the oblique orientation of the ACL and the constraint provided by the ACL at various tibial rotation and adduction positions. The purposes of this study were to evaluate multiaxis muscle strength in ACL deficient and reconstructed knees and to gain insights into potential compensatory mechanisms adopted by the patients.
    Muscle strength in tibial internal-external rotation, abduction-adduction, and flexion-extension were investigated in 19 chronic ACL deficient, 18 acute ACL deficient, 21 ACL reconstructed, and 23 normal subjects. The strength ratios of flexion/extension, abduction/adduction, and internal/external rotation were determined for each subject and compared across the different populations.
    The chronic ACL deficient patients showed significantly lower strength ratio in internal/external rotation than that of the normal controls and acute ACL deficient subjects (P = 0.02), indicating a compensatory mechanism developed by the patients to unload the ACL and/or to avoid unstable knee positions. For ACL reconstructed patients, the internal/external rotation strength ratio became closer to their counterparts in normal controls than that of chronic ACL deficient patients, presumably reflecting the reduced need for compensation after reconstruction. Furthermore, compared with strength reduction in knee extension, reductions in tibial rotation and abduction strength following ACL reconstruction were less severe and more easy to recover.
    A better understanding of changes in multiaxis muscle strength and the associated compensatory mechanism will help us evaluate treatment outcome more accurately and develop more effective treatment modalities with focus on muscles that help protect and unload the ACL.
    PMID: 11782640 [PubMed - as supplied by publisher]
  • Stiffness, viscosity, and upper-limb inertia about the glenohumeral abduction axis.

    Journal of orthopaedic research : official publication of the Orthopaedic Research Society 2000 Jan

    Authors: Zhang LQ,
    Abstract
    To evaluate the dynamic properties of the shoulder and understand how they are controlled by the central nervous system, glenohumeral-joint stiffness and viscosity and upper-limb inertia were quantified under various levels of muscle contraction in seven healthy human subjects. Through a cast attachment, the upper limb was perturbed in a precise pattern by a computer-controlled servomotor to manifest the dynamic properties of the joint. The recorded joint position and torque were used to estimate joint stiffness and viscosity and upper-limb inertia. With moderate muscle contraction, the stiffness and viscosity increased several fold. A stiffer shoulder joint associated with stronger muscle contraction made the shoulder more stable and protected it from potential injuries during strenuous tasks. Joint viscosity, especially the stronger viscous damping associated with more strenuous contraction, smoothed shoulder movement and stabilized the joint. From the control viewpoint, the glenohumeral joint responded to the central nervous system more quickly with increasing muscle contraction, which was useful during strenuous tasks. On the other hand, the central nervous system controlled stiffness and viscosity synchronously so that it dealt with only a nearly constant damping ratio of the joint over various levels of contraction, which simplified its task substantially. This approach quantified the dynamic and static properties of the shoulder under various levels of contraction more accurately and completely than a manual test, and it can potentially be used to evaluate changes in these properties caused by musculoskeletal injuries and their surgical treatments.
    PMID: 10716284 [PubMed - as supplied by publisher]
  • Spur reformation after arthroscopic acromioplasty.

    Arthroscopy : the journal of arthroscopic & related surgery : official publication of the Arthroscopy Association of North America and the International Arthroscopy Association 1999 Oct

    Authors: Anderson K,
    Abstract
    Rotator cuff pathology has been associated with a hooked acromial morphology. Impingement syndrome has traditionally been considered to be the result of bony encroachment into the subacromial space. This report of a spur recurrence after acromioplasty presents evidence that acromial morphology may be a reactive change attributable to primary rotator cuff insufficiency.
    PMID: 10524832 [PubMed - as supplied by publisher]
  • Effect of joint compression on inferior stability of the glenohumeral joint.

    Journal of shoulder and elbow surgery / American Shoulder and Elbow Surgeons ... [et al.]

    Authors: Warner JJ,
    Abstract
    To determine the relative importance of negative intraarticular pressure, capsular tension, and joint compression on inferior stability of the glenohumeral joint we studied 17 fresh, normal adult cadaver shoulders using a "3 degrees of freedom" shoulder test apparatus. Translations were measured in intact and vented shoulders while a 50-N superior and inferior directed force was applied to the shoulder. Three different joint compressive loads (22 N, 111 N, 222 N) were applied externally. Tests were performed in 3 positions of humeral abduction in the scapular plane (0 degree, 45 degrees, 90 degrees) and in 3 positions of rotation (neutral, maximal internal, and maximal external). After tests of the intact and vented shoulder, the glenohumeral ligaments were sectioned and tests were repeated. With minimal joint compression of 22 N, negative intraarticular pressure and capsular tension limited translation of the humeral head on the glenoid. Increasing the joint compressive load to 111 N resulted in a reduction of mean inferior translation from 11.0 mm to 2.0 mm at 0 degree abduction, from 21.5 mm to 1.4 mm at 45 degrees abduction, and from 4.5 mm to 1.2 mm at 90 degrees abduction. With a compressive load of 111 N, venting the capsule or sectioning of glenohumeral ligaments had no effect on inferior stability. Clinical relevance: Glenohumeral joint compression through muscle contraction provides stability against inferior translation of the humeral head, and this effect is more important than negative intraarticular pressure or ligament tension.
    PMID: 10077793 [PubMed - as supplied by publisher]
  • Articular contact patterns of the normal glenohumeral joint.

    Journal of shoulder and elbow surgery / American Shoulder and Elbow Surgeons ... [et al.]

    Authors: Warner JJ,
    Abstract
    The purpose of this study was to determine the articular contact patterns of the normal glenohumeral joint, and to correlate these findings with cartilage and subchondral bone architecture. We studied 10 normal shoulders of cadavers. We removed all soft tissues except the joint capsule and rotator cuff and then placed the shoulders on a testing apparatus that allowed freedom of translation in three planes. After the humerus was placed in a neutral position of rotation, articular contact patterns were measured with specially prepared prescale Fuji film so that it could be inserted between the joint surfaces. Articular contact was analyzed with 222 and 444 N of joint compressive load, and the humerus was positioned in scapular plane abduction of 0 degree, 45 degrees, and 90 degrees. The contact patterns were then digitized to determine percentage contact of the humeral head on the glenoid. We studied 12 additional cadaver shoulders with fine microradiographs and histologic techniques after we sectioned the glenoids in the anterior-posterior and superior-inferior planes. We then analyzed articular and subchondral architecture. We found that when the shoulder was adducted the contact area of the humeral head on the glenoid was limited to the anatomic region of the central glenoid known as the "bare area." This was histologically and radiographically an area of cartilage thinning and increased subchondral bone density. As the shoulder was abducted the articular congruity and percentage contact area increased. We concluded that there was a slight articular mismatch with the shoulder adducted in the normal shoulder. Histologic and radiographic studies suggested that the central bare area region of the glenoid was a region of increased compressive loading. As the shoulder was abducted the joint became more congruent and thus the contact area of the humeral head on the glenoid increased.
    PMID: 9752648 [PubMed - as supplied by publisher]
  • Neurovascular problems in the forearm, wrist, and hand.

    Clinics in sports medicine 1998 Jul

    Authors: Nuber GW,
    Abstract
    This article attempted to summarize the most common neurovascular injuries of the upper extremity, particularly the forearm, wrist, and hand. Although these injuries are rarely encountered in athletes, their pathology and treatment must be understood by the treating physician. Failure to recognize these injuries in a timely manner can lead to delay in diagnosis and weeks or months of lost participation by the athlete. The sports medicine physician must be aware of the potential risk for injury to the neurovascular structures, particularly in the athlete exposed to repetitive use or impact of the upper extremity. Timely recognition, diagnosis, and treatment will avoid the potential risk for permanent injury.
    PMID: 9700421 [PubMed - as supplied by publisher]
  • Acromioclavicular Joint Injuries and Distal Clavicle Fractures.

    The Journal of the American Academy of Orthopaedic Surgeons 1997 Jan

    Authors: Nuber MK,
    Abstract
    The acromioclavicular joint is commonly affected by traumatic and degenerative conditions. Most injuries are due to direct trauma, such as a fall on the shoulder. Six types of acromioclavicular sprains and three types of distal clavicle fractures have been described in adults. Although there is general agreement on treatment of type I, II, IV, V, and VI acromioclavicular injuries, the treatment of type III injuries remains controversial. Studies have shown no distinct advantage for surgical reconstruction over nonoperative treatment. Because type II distal clavicle fractures are prone to nonunion, operative fixation may be advisable to avoid this complication.
    PMID: 10797203 [PubMed - as supplied by publisher]
  • Intraarticular fibrous nodule as a cause of loss of extension following anterior cruciate ligament reconstruction.

    Arthroscopy : the journal of arthroscopic & related surgery : official publication of the Arthroscopy Association of North America and the International Arthroscopy Association 1992

    Authors: Marzo JM,
    Abstract
    Loss of motion is a well-known complication following anterior cruciate ligament (ACL) reconstruction. We have found that loss of extension is more disabling than loss of flexion, and is a more common problem following arthroscopic assisted ACL reconstruction. We are reporting on a group of 21 patients who have developed restricted knee extension following ACL reconstruction utilizing either the central one-third of the patellar ligament or the hamstring tendons as an autogenous graft. The patients presented at an average of 4 months postoperatively with a clinical syndrome of loss of extension associated with pain at terminal extension, crepitus, and grinding with attempted extension beyond their limit. The consistent finding at arthroscopy was a fibrous nodule occupying the intercondylar notch, varying in size from 1 x 1 to 2 x 3 cm, and presenting a mechanical block to full extension. It appears that anterior placement of the graft, particularly on the tibia, results in injury to the graft and subsequent nodule formation. Removal of the nodule resulted in improvement of an average preoperative loss of extension of 11 degrees, to 3 degrees at surgery, and 0 degrees at 1 year follow-up. The average side-to-side difference in terminal extension at final examination, using the uninvolved limb for comparison, was 3 degrees. Histology was available for review on 19 of the 21 patients operated on. The consistent microscopic finding within the nodule was the presence of disorganized dense fibroconnective tissue that, with time, underwent modulation to fibrocartilage. It is postulated that this occurs in response to compressive loading of the nodule.
    PMID: 1550639 [PubMed - as supplied by publisher]

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